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ESRS 50th Anniversary JSR Special Issue Webinar

September 5 @ 3:00 pm - 4:30 pm UTC+0

In celebration of 50 years of ESRS, the Journal of Sleep Research recently published a special edition commemorating this. It features a collection of 21 articles from well-known European clinicians and researchers from the fields of sleep medicine and sleep research.

This webinar will feature presentations from key several contributors of the special issue, including:

Alexander Borbély: Origin and relevance of the two-process model of sleep regulation
The two-process model serves as a major conceptual framework in sleep science. He will describe how animal experiments aimed at exploring circadian oscillators led to the recognition of a sleep-wake-dependent process. Its interaction with a circadian process provided the basis of the two-process model. It was proposed that the emergence of sleep homeostasis provided an escape from the rigid control imposed by the circadian pacemaker.

Kerstin Hödlmoser: Memory consolidation during sleep
Sleep has been demonstrated to play a significant role for memory consolidation and sleep scientists have started unravelling its underlying neurophysiological mechanisms. However, sleep remains a multidimensional phenomenon, and many questions remain left open for future research. In this presentation she will highlight the developmental trajectory of sleep-dependent memory consolidation processes, from their development in childhood to their potential impairments in ageing, and the nature and extent of our capabilities for information processing, learning, and memory reinforcement during sleep.

Dirk Pevernagie: OSA or OSAS? a half-century of progressing insight
Obstructive sleep apnea (OSA) and OSA syndrome (OSAS) were fully described by European researchers and investigators from Stanford university in the early seventies. OSAS was defined as the presence of obstructive respiratory events during sleep, as shown on polysomnography, in association with suggestive symptoms, e.g. excessive daytime sleepiness, and cardiometabolic complications such as hypertension. The operational definition thus seemed straightforward: OSAS = OSA + symptoms and signs. However, this equation proves problematic. Its application in clinical research and medical practice has produced equivocal outcomes. Nowadays we know that the pathophysiology of OSA is not tightly linked to clinical manifestations. OSAS appears to be a complex and heterogeneous disorder. Not all subjects with OSA require treatment, and for those who should be treated a personalized approach is recommended.

Birgit Högl: RBD: Past, present and future


September 5
3:00 pm - 4:30 pm UTC+0
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